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CDKN1A — RNF19A

Text-mined interactions from Literome

Kim et al., J Cell Physiol 2002 : Expression of dominant negative p38MAPK also blocked arsenite induced p21 ( CIP1/WAF1 ) expression
Ussat et al., Biochem Biophys Res Commun 2002 (Necrosis) : The use of specific pharmacological inhibitors revealed a partial involvement of p38 mitogen activated protein kinase in the upregulation of p21 ( WAF1/Cip1 )
Roy et al., Circ Res 2003 (Hyperoxia) : The molecular signature response to perceived hyperoxia included ( 1 ) induction of p21 , cyclin D1, cyclin D2, cyclin G1, Fos related antigen-2, and transforming growth factor-beta1, ( 2 ) lowered telomerase activity, and ( 3 ) activation of transforming growth factor-beta1 and p38 mitogen activated protein kinase
Zhang et al., Free Radic Biol Med 2003 : PD98059, an inhibitor of ERK, and SB202190, an inhibitor of p38 , inhibited vanadate induced cell growth arrest, upregulation of p21 and cdc2, and degradation of cdc25C
Seo et al., J Biol Chem 2004 : UV-induced p38 activation by Gbetagamma was blocked by overexpression of dominant negative p21 activated kinase (PAK) interacting exchange factor beta ( betaPix ), and wild type betaPix stimulated the UV-induced p38 activation, which was blocked by N17 Cdc42
Moon et al., Biochem Biophys Res Commun 2004 : In conclusion, we report the novel finding that transcription factor Sp1 that is involved in the p38MAPK mediated control of p21WAF1 regulation on VSMC in response to PDTC has now been identified
Halawani et al., Oncogene 2004 (Chondrosarcoma) : In addition, p38 inhibitors induced increased transcription of the cell-cycle inhibitor p21 ( waf1/cip1 )
Nuntharatanapong et al., Am J Physiol Heart Circ Physiol 2005 : Moreover, inhibition of JNK with SP-600125 or dominant negative MKK7 inhibited only p21Cip1/Waf1 induction, whereas the p38 MAPK inhibitor SB-203580 or dominant negative MKK4 inhibited both p21Cip1/Waf1 and p53 induction
Schepers et al., Exp Hematol 2005 (Acute Disease...) : Overexpression of cytoplasmic p21 inhibited the enhanced p38 phosphorylation after HSP27 RNAi, suggesting a predominant anti-apoptotic role of p21 over HSP27
Nicke et al., Mol Cell 2005 (Cell Transformation, Neoplastic...) : p38 MAPK activation is essential for Ras induced p21 ( WAF1/CIP1 ) upregulation and cell cycle arrest
Moon et al., Biochem Biophys Res Commun 2006 : In conclusion, we report that the transcription factor Sp1 involved in the p38MAPK mediated control of p21WAF1 regulation on VSMC in response to BITC has now been identified
Schultze et al., Cell Res 2012 : Markedly, loss of p38a leads to downregulation of p21 , and decreased activation of the p21 target Rb, both of which are important regulators of erythroblast enucleation