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BAX — HK1

Text-mined interactions from Literome

Rathmell et al., Mol Cell Biol 2003 : Nevertheless, expression of Glut1 and HK1 promoted increased cytosolic NADH and NADPH levels relative to those of the control cells upon growth factor withdrawal, prevented activation of Bax , and promoted growth factor independent survival
Gall et al., Kidney Int 2011 (Disease Models, Animal...) : Compared with control, adenoviral mediated HK I or II overexpression improved cell survival following stress, but did not prevent GSK3ß or Bax activation, improve ATP content, or reduce mitochondrial fragmentation