Crouch et al., J Cell Biol 2001
:
Thus, in Swiss 3T3 cells the G protein coupled thrombin receptor can potentiate the EGF tyrosine kinase receptor response when activated by EGF, and this appears to be due to the subcellular concentration of the receptor with downstream effectors and not to the overall ability of EGF to induce receptor transphosphorylation
Muroya et al., Biochim Biophys Acta 1992
(Adrenal Gland Neoplasms...) :
Tyrosine kinase activity of gp140trk , a constituent of NGF receptor, is activated by NGF for much a longer period compared to the activation of EGF receptor autokinase activity by EGF Wiederholt et al., Invest Ophthalmol Vis Sci 1998
:
The signaling cascade after activation of a tyrosine kinase receptor by EGF is differently modulated in trabecular meshwork and ciliary muscle