◀ Back to ATP5O

ATP5O — FGFR3

Text-mined interactions from Literome

Palacios et al., Am J Physiol Heart Circ Physiol 2004 : Ovariectomy eliminated sex differences in Na ( + ) -K ( + ) -ATPase function, and chronic in vivo hormone replacement with 17beta-estradiol restored the ACh effect on Na ( + ) -K ( + ) -ATPase
Ando et al., J Neurochem 1986 : Ganglionic Na+, K+-ATPase activity was greatly stimulated in the presence of NE, but not by ACh
Ando et al., Brain Res 1986 : On the other hand, ganglionic Ca2+, Mg2+-ATPase activity was greatly stimulated by either GMF or ACh
Kanyicska et al., Pol J Pharmacol Pharm 1985 (Catalepsy) : Inhibition of Na/K ATPase by ouabain led to an increase of striatal acetylcholine (Ach) release, which was enhanced by Met-enkephalin
Aperia et al., Acta Physiol Scand 1984 : We have previously demonstrated that adrenal corticoid hormones (ACH) induce NaK-ATPase activity in immature proximal tubular ( PT ) cells in rats
Vizi et al., J Physiol (Paris) 1982 : 5. The stimulation of membrane ATPase ( by switching off K and its readmission ) results in an inhibition of both ACh and noradrenaline release evoked by axonal stimulation
Elaev et al., Biull Eksp Biol Med 1983 : The latter fact evidences the ACh induced synthesis of activating factor and inhibition of Na, K-ATPase synthesis