◀ Back to GRAP2
CSF2 — GRAP2
Text-mined interactions from Literome
Bozinovski et al., J Biol Chem 2002
(MAP Kinase Signaling System) :
LPS markedly activated Akt and Erk1/2, but not
p38 , in a
GM-CSF dependent manner in direct temporal association with NFkappaB and AP-1 activation
Senokuchi et al., J Biol Chem 2005
:
GM-CSF induced p38 MAPK phosphorylation was also inhibited by farnesyl transferase inhibitor or geranylgeranyl transferase inhibitor, and farnesyl pyrophosphate and geranylgeranyl pyrophosphate prevented the suppression of
GM-CSF induced
p38 MAPK phosphorylation by statins
Scicchitano et al., Blood Cells Mol Dis 2008
(MAP Kinase Signaling System) :
In addition, these results suggest that IL-6, IL-11,
GM-CSF , G-CSF and Activin A are similarly
regulated by
p38 and NF-kappaB and that the MEK1, JNK and PKC pathways appear to play a more limited role in modulating cytokine expression in HBMSC
Souza-Fonseca-Guimaraes et al., J Biol Chem 2013
(MAP Kinase Signaling System) :
IFN-? and
GM-CSF production
requires NF-?B and STAT3 activation as well as Erk dependent mechanisms for IFN-? and
p38 signaling for GM-CSF
Aoshiba et al., J Immunol 1999
:
Granulocyte-macrophage
CSF and anti-Fas Ab, which altered the rate of apoptosis, did not
affect phosphorylation and activation of
p38-MAPK