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FASLG — IL6

Text-mined interactions from Literome

Arnold et al., J Immunol 1999 : Stimulation of the cells with IL-6 , IL-10, IL-12, TGF-beta1, and GM-CSF did not modulate the constitutive FasL expression, but IFN-gamma mediated FasL up-regulation was significantly diminished by IL-10 and TGF-beta1, respectively
Atsumi et al., J Exp Med 2002 (Arthritis, Experimental) : The inhibitory effect of IL-6 on Fas ligand expression during activation induced cell death ( AICD ) was augmented in gp130 ( F759/F759 ) T cells in a manner dependent on the tyrosine residues of gp130 required for signal transducer and activator of transcription 3 activation
Duan et al., Clin Cancer Res 2004 (Osteosarcoma) : The purpose of these studies was to determine whether alkylating agents affect the expression of Fas ligand (FasL) and whether interleukin 12 enhances the sensitivity of human OS cells to alkylating agents
Turner et al., J Soc Gynecol Investig 2004 : Fas ligand stimulated IL-6 secretion ( 3.06 +/- 0.38 fmol/mg wet weight per 3 hours, P < .05 ) ... Prostanoids, NSAIDs, and the Fas ligand regulate placental IL-6 secretion
Farley et al., J Invest Dermatol 2006 (Dermatitis...) : In HaCaT cells and in reconstructed human epidermis ( RHE ), FasL triggered a NF-kappaB dependent mRNA accumulation of inflammatory cytokines ( tumor necrosis factor-alpha, IL-6 , and IL-1beta ), chemokines ( CCL2/MCP-1, CXCL1/GROalpha, CXCL3/GROgamma, and CXCL8/IL-8 ), and the adhesion molecule ICAM-1
Seitz et al., Shock 2011 (Inflammation...) : Fas ligand stimulation further enhanced IL-6 and suppressed IL-10 release in AMFs after trauma.The results indicate that the Fas/FasL system is activated after chest trauma, and FasL is associated with the inflammatory response after lung contusion
Hu et al., Mol Med Report 2008 : SOCS1 silencing in DCs could prevent immune tolerance by inhibiting Fas and Fas-L expression, induced by an increase in IL-12p70 and IL-6 production
von Rossum et al., J Biol Chem 2011 (Inflammation) : IL-1, but not IL-6 , inhibited expression of FasL , whereas IL-6, but not IL-1, increased c-FLIP ( S ) expression
Erwin et al., Arthritis Res Ther 2011 (Intervertebral Disc Degeneration) : Furthermore, NP cells treated with NCCM in the presence of IL-1ß+Fas-L down-regulate the expression of IL-6 by almost 50 %
Greene et al., J Interferon Cytokine Res 1997 : We have investigated the role of interleukin-6 (IL-6) in the induction of major histocompatibility complex ( MHC ) -unrestricted cytotoxicity, as well as granzyme B, perforin, and Fas ligand gene expression, following mouse T lymphocyte activation with anti-CD3 monoclonal antibody ( mAb )
Ben Amor et al., Exp Hematol 1998 : Together, our data support the notion that the decrease in IL-3 induced histamine and IL-6 production by splenocytes pretreated with anti-CD3 is mediated , at least in part, by Fas/FasL interactions, suggesting that the activity of extramedullary myeloid precursor cells can be modulated by molecules involved in apoptosis
Ayroldi et al., Blood 1998 : We found that IL-6 reduced the level of TCR/CD3 induced apoptosis and the expression of Fas/FasL , yet failed to inhibit IL-2 production