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ALB — APOB
Text-mined interactions from Literome
Janisch et al., Free radical research 2004
:
The data show that flavonoid intestinal and hepatic metabolism have profound effects on ability to inhibit
LDL oxidation and a lesser but significant
effect on binding to serum
albumin
Jayaraman et al., Biochemistry 2007
:
Consequently, oxidation alone may prevent rather than promote LDL fusion, suggesting that additional factors, such as
albumin mediated removal of lipid peroxidation products and/or
LDL binding to arterial proteoglycans, facilitate fusion of oxidized LDL in vivo
Song et al., Dig Liver Dis 2009
(Cholestasis, Intrahepatic...) :
Biochemical analysis revealed intrahepatic cholestasis,
delayed switch of AFP to
albumin , and elevated triglyceride, total cholesterol and
LDL-cholesterol together with reduced HDL-cholesterol
Karmanskiĭ et al., Biull Eksp Biol Med 1977
:
It was shown that the action of phospholipase A2 on
low density serum lipoproteins (LDL) in the
presence of serum
albumin led to decrease in the floatation coefficient
Shireman et al., Proc Natl Acad Sci U S A 1977
:
Unlabeled LDL and apo
B-albumin complex both competitively
inhibited the binding of ( 125 ) I-labeled apo B and ( 125 ) I-labeled
LDL to fibroblasts
Gordon et al., J Agric Food Chem 2010
:
Bovine serum
albumin ( BSA )
increased the oxidative stability of
LDL in the presence of chlorogenic acid
Horiuchi et al., J Biol Chem 1985
:
By contrast, the binding of
125I-acetyl-LDL to the cells was
affected neither by unlabeled
f-Alb nor by the antibody raised against the f-Alb receptor
Dresel et al., EMBO J 1985
(Carcinoma 256, Walker) :
Polyanionic competitors and maleylated bovine serum
albumin inhibited the binding of
acetyl-LDL effectively
Crane et al., Mol Cell Biochem 1983
(Liver Neoplasms, Experimental) :
Lipoprotein synthesis is stimulated by fatty acids and is
inhibited by
albumin and other macromolecules
Ji et al., Zhongguo Zhong Xi Yi Jie He Za Zhi 1993
(Kidney Failure, Chronic...) :
Serum levels of TC,
LDL-c , apo B and apo B/apo A1 dropped down and HDL-c, apo A1, albumin,
pro-albumin and fibrinectin
increased during the follow-up period ( P < 0.05 ) in the treated patients