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BDNF — JUN
Text-mined interactions from Literome
Ruiz-León et al., J Neurochem 2001
(Neuroblastoma) :
These results suggest that activation of the APP promoter by
BDNF is largely
independent of PKC and
AP-1
Yamagishi et al., Brain Res Mol Brain Res 2003
(Potassium Deficiency) :
Comparison of inhibitory
effects of
brain derived neurotrophic factor and insulin-like growth factor on low potassium induced apoptosis and activation of p38 MAPK and
c-Jun in cultured cerebellar granule neurons ...
BDNF and IGF-1
suppressed the activation of p38 and
c-Jun , but not of c-Jun N-terminal kinase (JNK), caused by lowering the potassium concentration
Wu et al., Neurobiol Dis 2012
:
We found that
BDNF dependent
c-Jun expression and nuclear translocation required prior phosphorylation of extracellular signal regulated kinase ( ERK ) 1/2, but not Akt
Gaiddon et al., J Neurochem 1996
(Second Messenger Systems) :
Here we show that
BDNF stimulates
AP1 binding activity in primary cerebellar neurons ... In summary, we show that
BDNF stimulates
AP1- and CRE dependent transcription through a mechanism that is distinct from the cAMP- and Ca ( 2+ ) -dependent pathways in CNS neurons
Sano et al., Biochem Biophys Res Commun 1996
:
These results indicate that nascent
AP-1 is not
required for the activation of
BDNF gene, leading to the notion that the BDNF gene can be activated by Ca2+ signals as an immediate early gene
Courtney et al., J Neurosci 1997
:
The cells express TrkB protein and show elevated c-Fos and
c-Jun levels in
response to
BDNF
Hsieh et al., Exp Neurol 1998
(Body Weight...) :
BDNF restores the expression of
Jun and Fos inducible transcription factors in the rat brain following repetitive electroconvulsive seizures