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NEUROD4 — NEUROG1

Text-mined interactions from Literome

Geling et al., Development 2004 : This involves selective suppression of both neurogenin1 (ngn1) and coe2 mRNA expression in a process that is independent of Notch signalling, and where inhibition of either ngn1 or coe2 expression is sufficient to prevent neuronal differentiation across the midbrain-hindbrain boundary
Kim et al., Cell Mol Neurobiol 2004 : Neurogenin1 is sufficient to induce neuronal differentiation of embryonal carcinoma P19 cells in the absence of retinoic acid
Turnley et al., Pediatr Endocrinol Rev 2005 : GH and SOCS2 have recently been shown to regulate neuronal differentiation by controlling expression of a neurogenic transcription factor, Neurogenin-1
Gasa et al., Differentiation 2008 : Interestingly, in addition to NeuroD1, Neurog3 also induced both NeuroD2 and NeuroD4 gene expression
Reyes et al., J Neurosci 2008 (Deafness) : Transient expression of Neurog1 promoted widespread neuronal differentiation in vitro ; when supplemented with brain derived neurotrophic factor (BDNF) and glial cell line derived neurotrophic factor ( GDNF ), 75 % of ES cell derived neurons attained a glutamatergic phenotype after 5 d in vitro
Quiñones et al., Dev Biol 2010 : The bHLH transcription factor Neurog1 ( Ngn1, Neurod3, neurogenin 1 ) is involved in neuronal differentiation and cell-type specification in distinct regions of the developing nervous system
Velkey et al., Dev Dyn 2013 : Results : Transient expression of Neurog1 in ESC was sufficient to initiate neuronal differentiation , and produced neuronal subtypes reflecting its expression pattern in vivo
Olson et al., Mol Cell Neurosci 1998 (Choristoma...) : GSK3beta, which inhibits NeuroD function in vivo, blocks both Ngn1 induced and NeuroD induced neuronal differentiation, while Notch signaling inhibits only Ngn1 induced neuronal differentiation , confirming that NeuroD is downstream of Ngn1 and insensitive to Notch inhibition