Gene interactions and pathways from curated databases and text-mining

◀ Back to EGF

EGF — FGF7

Text-mined interactions from Literome

Hienola et al., Mol Cell Neurosci 2004 : Further, our in vitro studies show that exogenously added HB-GAM inhibits formation and growth of FGF-2, but not EGF , stimulated neurospheres, restricts the number of nestin positive neural stem cells, and inhibits FGF receptor phosphorylation
Horiguchi et al., Endocrine 2004 : Basic FGF and EGF did not modulate changes in VEGF-A mRNA levels after 4 and 24 h of treatment, but EGF increased VEGF-A significantly ( p < 0.05 ) after 96 h of treatment
Fiorelli et al., J Clin Endocrinol Metab 1989 (Prostatic Hyperplasia) : EGF binding was inhibited by excess EGF, but not by insulin, proinsulin, fibroblast growth factor , or insulin-like growth factors I and II
Gospodarowicz et al., J Cell Physiol 1977 : With chondrocytes, both FGF and EGF are mitogenic and FGF 's, but not EGF 's effect is potentiated by dexamethasone
Balk et al., Life Sci 1984 : While EGF alone at 100 ng/ml causes threefold multiplication at four days and brain FGF causes a sixfold increase, EGF acts synergistically with Sm-C/IGF-I, MSA/rIGF-II, insulin or proinsulin to cause 18-fold multiplication, and brain FGF acts synergistically with IGFs to cause 20-fold multiplication
Yamagata et al., Clin Exp Pharmacol Physiol 1995 (Cerebrovascular Disorders...) : EGF induced greater DNA synthesis in SHRSP and SHRSR astrocytes compared with WKY astrocytes, although FGF had little or no effect
Peehl et al., Growth Regul 1996 (Prostatic Neoplasms) : Previous studies indicate that keratinocyte growth factor (KGF) acts as a paracrine factor in the prostatic epithelium and epidermal growth factor (EGF) acts as an autocrine factor