◀ Back to MMP1
IL34 — MMP1
Text-mined interactions from Literome
Meisser et al., Mol Hum Reprod 1999
:
The aim of this study was to determine the
effects of tumour necrosis factor alpha (TNF),
interleukin-1 alpha (IL-1alpha), macrophage colony stimulating factor ( MCSF ) and transforming growth factor beta ( TGFbeta ) on the secretion of
matrix metalloproteinases ( MMP ) , human chorionic gonadotrophin ( HCG ) and fetal fibronectin ( fFN ) by purified first trimester cytotrophoblastic cells (CTB) in vitro
Ray et al., Arthritis Rheum 2003
(Osteoarthritis) :
Overexpression of SAF-1 in OA chondrocytes increased the expression of the MMP1 promoter, and interference of endogenous SAF-1 activity by antisense SAF-1 messenger RNA inhibited
interleukin-1 mediated
MMP1 promoter activity
Sun et al., Connect Tissue Res 2003
(Arthritis, Rheumatoid) :
Furthermore, the
induction of the
MMP expression and activities by
interleukin-1 was suppressed by the oscillatory shear
Abe et al., J Nat Prod 2010
:
The effects of these new compounds on
interleukin-1ß induced production of
matrix metalloproteinase-1 (MMP-1) in human dermal fibroblasts were examined
Gotoh et al., Connect Tissue Res 2013
:
Extracellular ß-NAD ( + ) inhibits
interleukin-1 induced
matrix metalloproteinase-1 and -3 expression on human gingival fibroblasts
Langdon et al., Arthritis Rheum 1997
(Arthritis, Rheumatoid) :
Oncostatin M stimulates monocyte chemoattractant protein-1- and
interleukin-1 induced
matrix metalloproteinase-1 production by human synovial fibroblasts in vitro
Dean et al., J Biol Chem 1999
:
We have previously shown that the pyridinyl imidazole SB 203580, which inhibits it, blocks the
interleukin-1 induction of cyclooxygenase-2 (COX-2) and
matrix metalloproteinase 1 and 3 mRNAs in fibroblasts