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ANG — PGF

Text-mined interactions from Literome

Miyamoto et al., Domest Anim Endocrinol 2005 : We propose that the increase in luteal BF may be induced by NO from large arterioles surrounding the CL, and simultaneously uterine or exogenous PGF2alpha directly increases ET-1 and Ang II secretion from endothelial cells of microcapillary vessels within the CL, thereby suppressing P secretion by luteal cells
Pan et al., BMC cell biology 2010 (Atherosclerosis) : In the current study, whether Ang II could regulate PlGF expression, and the effect of PlGF on cell proliferation, was investigated in human vascular endothelial cells ( VECs ) and smooth muscle cells ( VSMCs ) ... Our results showed for the first time that Ang II could induce the gene expression and protein production of PlGF in VECs and VSMCs, which might play an important role in the pathogenesis of vascular inflammation and atherosclerosis
Anton et al., Reproductive biology and endocrinology : RB&E 2010 (Pre-Eclampsia) : We evaluated the effects of angiotensin II (Ang II) and angiotensin- ( 1-7 ) [ Ang- ( 1-7 ) ] on the release of VEGF, PLGF , sFlt1, and sEng from placental chorionic villi ( CV )
Virdis et al., Eur Heart J 2012 : In WT, Ang II increased COX-1 and decreased COX-2 expression, and enhanced the vascular release of 6-keto-PGF1a which was prevented by COX-1 blockade
Magness et al., J Clin Invest 1985 : ANG II increased 6-keto-PGF1 alpha production 107 +/- 20 % and 92 +/- 16 % in P and early uterine arteries only ; the threshold dose was between 5 X 10 ( -11 ) and 5 X 10 ( -9 ) M ANG II
Corriu et al., J Cardiovasc Pharmacol 1995 : We conclude that in both rat resistance and conductance vessels, ANG II induces vasoconstriction through activation of AT1 receptors which are selectively blocked by losartan at nanomolar concentrations and that at micromolar concentrations, losartan may also block the vascular TXA2/PGF2 alpha (TP) receptor