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CA2 — PLCB3

Text-mined interactions from Literome

Hwang et al., Mol Cells 2005 : Phospholipase C-beta3 mediates the thrombin induced Ca2+ response in glial cells ... These results suggest that activation of PLC-beta3 by pertussis toxin-sensitive G proteins is responsible for the transient [ Ca2+ ] i increase in response to thrombin, whereas the delayed [ Ca2+ ] i increase may be due to activation of some other PLC, such as PLC-beta4, acting via PTx-insensitive G proteins
Garic-Stankovic et al., Alcohol Clin Exp Res 2005 (Disease Models, Animal) : Ethanol triggers neural crest apoptosis through the selective activation of a pertussis toxin-sensitive G protein and a phospholipase Cbeta dependent Ca2+ transient
Park et al., J Biol Chem 1993 : A nearly saturating concentration of beta gamma produced 2.5-, 4-, 8.5-, and 2-fold increases in PLC-beta 1, PLC-beta 2, PLC-beta 3 , and PLC-delta 1 activity, respectively, and no activation of PLC-gamma 1, in the presence of 0.2 microM free Ca2+