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CALM2 — FGFR3

Text-mined interactions from Literome

Malomouzh et al., J Neurochem 2007 (Calcium Signaling...) : It seems that a substantial part of the H-effect can be physiologically regulated by this negative feedback loop, i.e., by NO from muscle fiber ; there is apparently also Ca ( 2+ ) - and calmodulin dependent regulation of ACh non-quantal release in the nerve terminal itself, as calmidazolium inhibition of the calmodulin led to a doubling of the resting H-effect
Ribeiro et al., Brain Res Bull 1986 : We observed that in cortical slices of brain, the TsTX induced release of ACh was inhibited in the presence of the calmodulin inhibitors trifluoperazine, W-7 and prenylamine, but not by trifluoperazine sulphoxide