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Caspase-1 activity is required for neuronal differentiation of PC12 cells : cross-talk between the caspase and calpain systems
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After treatment with the neurotoxicant trimethyltin ( TMT ), differentiated M3GFP-BDNF exhibit an attenuation of cellular damage which correlates with a significant activation of MAPK and PI3K/Akt signaling and delayed activation of death signals, while on M3GFP, TMT induces a significant reduction of cell survival, neuronal differentiation and concomitant earlier activation of cleaved caspase-3