◀ Back to NOS3
ARG1 — NOS3
Text-mined interactions from Literome
Baggio et al., J Pharmacol Exp Ther 1999
:
We conclude that
arginase plays a significant role in the regulation of
nitric oxide synthase mediated NANC relaxation in the IAS
Liu et al., Sheng Li Xue Bao 2002
(Cardiomegaly) :
L-Arg also
increased the gene expression of
eNOS
White et al., Hypertension 2006
:
Moreover,
arginase inhibition
restores endothelial NOS signaling and l-arginine responsiveness in old rat aorta
Sasaki et al., Am J Physiol Lung Cell Mol Physiol 2007
(Hypertension, Pulmonary...) :
Roles of accumulated endogenous
nitric oxide synthase inhibitors,
enhanced arginase activity, and attenuated nitric oxide synthase activity in endothelial cells for pulmonary hypertension in rats
Liu et al., Amino Acids 2012
(Birth Weight) :
In conclusion, this study demonstrated that dietary
Arg or NCG supplementation may
affect microRNAs ( miR-15b, miR-222 ) targeting VEGFA and
eNOS gene expressions in umbilical vein, so as to regulate the function and volume of the umbilical vein, provide more nutrients and oxygen from the maternal to the fetus tissue for fetal development and survival, and enhance the reproductive performance of sows
Shin et al., The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology 2011
:
In HUVECs, inhibition of
arginase activity by CLE reciprocally
increased NOx production through enhancement of
eNOS dimer stability without any significant changes in the protein levels of eNOS and arginase II expression
Prieto et al., Placenta 2011
(Anoxia) :
We studied in human umbilical vein endothelial cells ( HUVEC ) whether hypoxia reduced NO synthesis results from increased
arginase mediated l-arginine metabolism and changes in subcellular localization of
eNOS and arginase-2