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MSN — RAC1
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Jeon et al., J Biol Chem 2005
:
Chloride conductance is required for the protein kinase A and
Rac1 dependent phosphorylation of
moesin at Thr-558 by KCl in PC12 cells ...
Moesin phosphorylation at Thr-558 was increased after 2 min of KCl treatment, peaked at 5 min, and returned to the basal level by 60 min. KCl also
activated Rac1 , but not RhoA, in PC12 cells, and KCl induced moesin phosphorylation was suppressed in dominant negative Rac1 ( N17 Rac1 ) -expressed cells ... These results suggest that the phosphorylation of
moesin at Thr-558 in PC12 cells by KCl treatment is PKA- and
Rac1 dependent and that KCl induced chloride conductance is involved in the activation of this signaling system
Rossy et al., Exp Cell Res 2007
(Carcinoma 256, Walker) :
We further show that endogenous ezrin and
moesin are phosphorylated and that activities of PI-3 kinase, Rho and
Rac , but not of Rho-kinase, are
required for this C-terminal phosphorylation
Jeon et al., Neurochem Int 2010
:
NGF induced
moesin phosphorylation is
mediated by the PI3K,
Rac1 and Akt and required for neurite formation in PC12 cells
Shaw et al., Mol Biol Cell 1998
:
An activated allele of RhoA, but not
Rac or CDC42Hs, was
sufficient to induce apical membrane/actin protrusions and localize radixin or
moesin into these structures in both Rat1 and NIH3T3 cells