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CSF3 — IL3

Text-mined interactions from Literome

Yamaguchi et al., Clin Exp Allergy 1992 : Both IL-3 and granulocyte-macrophage colony stimulating factor ( GM-CSF ) induced migration of purified normal basophils ( purity c. 80 % ) in a dose dependent fashion at picomolar concentrations, while granulocyte (G)-CSF , macrophage (M)-CSF, and IL-4 had no effect at all
Akahane et al., Arzneimittelforschung 1990 : The enhanced G-CSF production from NFSA cells may be due at least to interleukin-1 released from muroctasin stimulated macrophages
Lopez et al., J Cell Physiol 1990 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : Specificity studies under conditions that prevent receptor internalization showed that the binding of IL-3 , GM-CSF, and IL-5 was not inhibited by tumor necrosis factor (TNF)-alpha, IL-1 beta, interferon (IFN)-gamma, or G-CSF
Leizer et al., Blood 1990 : Cytokine regulation of colony stimulating factor production in cultured human synovial fibroblasts : I. Induction of GM-CSF and G-CSF production by interleukin-1 and tumor necrosis factor
Kataoka et al., Cancer Res 1990 (Leukemia, Myeloid, Acute) : 125I-G-CSF binding was not inhibited by interleukin-3 , granulocyte-macrophage CSF, or macrophage (M) CSF
Schipperus et al., Leuk Res 1990 (Myelodysplastic Syndromes) : Il-3 did not enhance the G-CSF activity in NBM cultures
Jiang et al., J Int Med Res 2010 (Alzheimer Disease...) : Levels of a7 nAChR protein were significantly increased and levels of interleukin-1ß , tumour necrosis factor-a and nuclear factor-?B ( NF-?B ) protein were significantly decreased in the brain of APP transgenic mice in response to G-CSF
Wang et al., Int J Oncol 1995 : We report that all-trans retinoic acid ( tRA ) synergizes with LPS to enhance the production of granulocyte colony stimulating factor ( G-CSF ) in PMA treated cells, whereas the production of granulocyte-macrophage CSF, interleukin 1-beta (IL-1-beta), and tumor necrosis factor-alpha (TNF-alpha) is minimally affected by tRA
Vellenga et al., Blood 1988 : Recombinant human interleukin-3 (IL-3) and GM-CSF primarily induce expression of the M-CSF gene and secretion of M-CSF, whereas bacterial lipopolysaccharide primarily induces expression of the G-CSF gene and secretion of G-CSF
Yanai et al., Leuk Res 1994 (Anemia) : IL-3 induced the production of IL-6, GM-CSF and G-CSF from the bone marrow cells of +/+ mice
Tsuji et al., Growth Factors 1994 : IL-3 induced expression of granulocyte-macrophage colony stimulating factor ( GM-CSF ) and granulocyte colony stimulating factor ( G-CSF ) in total bone marrow cells and a marrow adherent cell population ... IL-3 also induces GM- and G-CSF expression which in turn makes granulocyte-macrophage colony forming cells differentiate to granulocytes
Nemoto et al., Biol Pharm Bull 1994 : In the genes related to haematopoiesis, OL-2 induced G-CSF and GM-CSF, but SPG induced IL-3
Ogawa et al., Am J Hematol 1996 : IL-2, IL-3 , IL-4, IL-5, and IL-6 were unable to induce G-CSF production
Pajkrt et al., Blood 1997 (Endotoxemia) : In group 1, G-CSF significantly enhanced the release of tumor necrosis factor (TNF), interleukin-6 (IL-6), IL-8, IL-1 receptor antagonist (IL-1ra), and soluble TNF receptors