◀ Back to ABL1

ABL1 — SHC1

Pathways - manually collected, often from reviews:

• KEGG Chronic myeloid leukemia: ABL1/BCR → SHC1/SHC2/SHC3/SHC4 (protein-protein, activation)

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: SHC1 — ABL1 (physical association, affinity chromatography technology) Brehme et al., Proc Natl Acad Sci U S A 2009
• IRef Biogrid Interaction: SHC1 — ABL1 (physical association, affinity chromatography technology) Wisniewski et al., Blood 1999*
• IRef Biogrid Interaction: SHC1 — ABL1 (physical association, affinity chromatography technology) Puil et al., EMBO J 1994*

Text-mined interactions from Literome

Matsuguchi et al., J Biol Chem 1994 (Leukemia, Megakaryoblastic, Acute) : Shc phosphorylation in myeloid cells is regulated by granulocyte macrophage colony stimulating factor, interleukin-3, and steel factor and is constitutively increased by p210BCR/ABL
Owen-Lynch et al., J Biol Chem 1995 : Both IL-3 and v-Abl stimulated the tyrosine phosphorylation of SHC and GTPase activating protein