◀ Back to CDC6

CDC6 — TPP1

Text-mined interactions from Literome

Miller et al., Mol Cell Biol 2001 : Both binding to Cdc28p and Cdc28p dependent phosphorylation in the C-terminal region of Cln2p are independently required for efficient nuclear depletion of Cln2p , suggesting that this process may be physiologically regulated
Colomina et al., Cell cycle (Georgetown, Tex.) 2009 : However, Cdc28 did not properly accumulate at the bud tip during S phase in whi3Delta cells, and their elongation defects were suppressed by CLN2 overexpression, suggesting a positive function for Whi3 in a Cdk-cyclin dependent step required for apical growth
Yaglom et al., Mol Cell Biol 1996 : The phosphorylation defect in the ydj1 mutant was specific to Cln3, because no reduction in the phosphorylation of Cln2 or histone H1, which also requires p34CDC28 , was observed
Levine et al., Mol Cell Biol 1998 : Cln2 immunoprecipitated from cdc28-csr1 cells was completely defective in histone H1 kinase activity, due to defects in Cdc28 binding and activation by Cln2
Wu et al., J Biol Chem 1998 : Overexpression of Cln2p , but not Clb2p or Clb5p, causes a quantitative shift of Ste20p to the reduced mobility form, and this shift is dependent on Cdc28p activity