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FOS — POLDIP2

Text-mined interactions from Literome

Chen et al., Mol Carcinog 2000 : In the present study, we further examined the role of p38 in UVB induced AP-1 activation ... These results suggested, for the first time, that activation of p38 is required for UVB induced AP-1 activation in human keratinocytes
Oh et al., J Biol Chem 2001 : This Lf-induced AP-1 DNA binding activity was reduced by a p38 MAPK inhibitor
Qi et al., J Biol Chem 2007 (Breast Neoplasms) : Analyses of MAPK kinase 6 ( MKK6 ) -p38 fusion proteins showed that constitutively active p38alpha ( MKK6-p38alpha ) and p38gamma ( MKK6-p38gamma ) stimulates and inhibits c-Jun phosphorylation respectively, leading to a distinct AP-1 regulation
Shim et al., Planta Med 2008 (MAP Kinase Signaling System) : Moreover, inhibition of ERK, JNK and p38 by panduratin A resulted in decreased c-Fos expression and c-Jun phosphorylation induced by UV, which led to inhibition of activator protein-1 (AP-1) DNA binding activity
Persichini et al., Neurosci Lett 2010 : Moreover, we have observed that IL-1ß regulates the expression of Cp and Fpn genes through ( i ) p38 MAPK mediated activation of C/EBP transcription factor, ( ii ) ERK1/2-, JNK1- and partially p38 MAPK dependent activation of AP-1 , and through ( iii ) activation of NF-?B partially mediated by p38 MAPK
Huang et al., Toxicol Appl Pharmacol 2013 (Inflammation) : Moreover, LTA induced increases of ?B-DNA and AP-1-DNA binding activity were inhibited by p38 , JNK, and PI3-kinase inhibitors