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CD44 — FOS

Text-mined interactions from Literome

Foster et al., FASEB J 2000 : Taken together, our studies demonstrate that AP-1 proteins are a central regulatory component used by IL-1beta to modulate expression of CD44 during an inflammatory response in vascular smooth muscle cells and that transcription of CD44 by AP-1 proteins is enhanced by HMG-I(Y)
Mishra et al., J Biol Chem 2005 (Calcium Signaling) : Differential involvement of calmodulin dependent protein kinase II-activated AP-1 and c-Jun N-terminal kinase activated EGR-1 signaling pathways in tumor necrosis factor-alpha and lipopolysaccharide induced CD44 expression in human monocytic cells ... Furthermore, TNF-alpha induced CD44 expression was regulated by AP-1 through the activation of the CaMK-II pathway, whereas LPS induced CD44 transcription was regulated specifically by Egr-1 through JNK activation
Vendrov et al., J Biol Chem 2010 (Atherosclerosis) : Thrombin induced CD44 expression is mediated by transcription factor AP-1 in a NADPH oxidase dependent manner
Smith et al., PloS one 2012 (Breast Neoplasms) : Cell specific CD44 expression in breast cancer requires the interaction of AP-1 and NF?B with a novel cis-element
Lamb et al., Mol Cell Biol 1997 (Cell Transformation, Neoplastic...) : We demonstrate that increased expression of CD44 in Fos- and EGF transformed cells is dependent upon AP-1
Fujii et al., J Immunol 1999 (Arthritis, Rheumatoid) : Briefly, we found that 1 ) rheumatoid synovial cells highly expressed CD44 ; 2 ) cross linking of CD44 markedly but transiently augmented VCAM-1 expression and its mRNA transcription much more than did IL-1beta and TNF-alpha ; 3 ) hyaluronan, especially when fragmented, also up-regulated VCAM-1 ; 4 ) CD44 activated the transcription factor AP-1 ; and 5 ) the integrin dependent adhesive function of RA synovial cells to T cells was also amplified by CD44 cross linking