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EGF — STK16

Text-mined interactions from Literome

Tartare et al., J Biol Chem 1991 : These results suggest that insulin-EGF chimeric receptor activation stimulates at least one serine/threonine kinase , which in turn phosphorylates the kinase-deficient EGF receptor
Cao et al., Science signaling 2009 : The precise mechanism whereby epidermal growth factor (EGF) activates the serine-threonine kinase Akt and the mammalian target of rapamycin (mTOR) complex 1 ( mTORC1 ) remains elusive
Ahn et al., J Biol Chem 1990 : Several separable EGF stimulated serine/threonine kinase activities were characterized in the preceding paper ( Ahn, N. G., Weiel, J. E., Chan, C. P., and Krebs, E.G. ( 1990 ) J. Biol. Chem. 265, 11487-11494 )
Saxena et al., Antioxid Redox Signal 2013 (Colonic Neoplasms...) : Inhibition of AR also prevented the epidermal growth factor induced phosphorylation of phosphatidylinositol 3-kinase (PI3K), serine/threonine kinase ( AKT ), c-Jun, c-Fos, PTEN, and FOXO3a, and deoxyribonucleic acid ( DNA ) -binding activity of AP-1
King et al., J Biol Chem 1986 : It has been suggested that EGF could indirectly activate a protein-serine/threonine kinase , protein kinase C, that can phosphorylate the EGF receptor at threonine 654