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CCNG1 — EPHB2

Text-mined interactions from Literome

O'Reilly et al., J Immunol 2009 : These results show that during mitogenic stimulation of T and B lymphocytes MEK/ERK signaling is critical for two distinct processes, cell survival, mediated ( at least in part ) through phosphorylation and consequent inhibition of Bim, and cell cycling , which proceeds independently of Bim inactivation
Wang et al., Blood 2013 (Leukemia, Myelomonocytic, Chronic) : The aberrant Nras ( G12D/+ ) HSC function is associated with hyperactivation of ERK1/2 in HSCs. Conversely, downregulation of MEK/ERK by pharmacologic and genetic approaches attenuates the cycling of Nras ( G12D/+ ) HSCs and prevents the expansion of Nras ( G12D/+ ) HSCs and myeloid progenitors