◀ Back to EGF

EGF — FGF2

Text-mined interactions from Literome

Santa-Olalla et al., J Neurobiol 1999 : Basic fibroblast growth factor promotes epidermal growth factor responsiveness and survival of mesencephalic neural precursor cells
Jaakkola et al., Prog Nucleic Acid Res Mol Biol 1999 : In fibroblasts syndecan-1 is induced by FGF-2 and in keratinocytes by epidermal growth factor (EGF) and keratinocyte growth factor (KGF)
Hienola et al., Mol Cell Neurosci 2004 : Further, our in vitro studies show that exogenously added HB-GAM inhibits formation and growth of FGF-2, but not EGF , stimulated neurospheres, restricts the number of nestin positive neural stem cells, and inhibits FGF receptor phosphorylation
Nouet et al., J Biol Chem 2004 : Ectopic expression of ATIP1 in eukaryotic cells leads to inhibition of insulin, basic fibroblast growth factor , and epidermal growth factor induced ERK2 activation and DNA synthesis, and attenuates insulin receptor autophosphorylation, in the same way as the AT2 receptor
Horiguchi et al., Endocrine 2004 : Basic FGF and EGF did not modulate changes in VEGF-A mRNA levels after 4 and 24 h of treatment, but EGF increased VEGF-A significantly ( p < 0.05 ) after 96 h of treatment
Smith et al., Endocrinology 2006 (MAP Kinase Signaling System) : Stimulation of Ishikawa cells and human endometrial biopsy explants with 100 nm iloprost ( a PGI analog ) rapidly activated ERK1/2 signaling and induced the expression of proangiogenic genes, basic fibroblast growth factor , angiopoietin-1, and angiopoietin-2, in an epidermal growth factor receptor (EGFR) dependent manner
Ahnstedt et al., Acta Physiol (Oxf) 2012 : bFGF ( 10 ng mL(-1) ) significantly increased the ET ( B ) mRNA level, and EGF ( 20 ng mL(-1) ) increased the ET ( A ) receptor protein
Fiorelli et al., J Clin Endocrinol Metab 1989 (Prostatic Hyperplasia) : EGF binding was inhibited by excess EGF, but not by insulin, proinsulin, fibroblast growth factor , or insulin-like growth factors I and II
Hicks et al., Biochem Biophys Res Commun 1989 : However, unlike phorbol esters, aFGF and bFGF inhibit EGF binding in protein kinase C-deficient cells ... In contrast to bombesin but like PDGF, neither aFGF nor bFGF act on the EGF receptor through a pertussis toxin-sensitive G protein
Gospodarowicz et al., J Cell Physiol 1977 : With chondrocytes, both FGF and EGF are mitogenic and FGF 's, but not EGF 's effect is potentiated by dexamethasone
Balk et al., Life Sci 1984 : While EGF alone at 100 ng/ml causes threefold multiplication at four days and brain FGF causes a sixfold increase, EGF acts synergistically with Sm-C/IGF-I, MSA/rIGF-II, insulin or proinsulin to cause 18-fold multiplication, and brain FGF acts synergistically with IGFs to cause 20-fold multiplication
Hayward et al., In Vitro Cell Dev Biol Anim 1995 (Aneuploidy) : In serum-free culture in plastic dishes epidermal growth factor (EGF) , transforming growth factor (TGF)-alpha, fibroblast growth factor (FGF) 1 ( aFGF ), and FGF 7 ( KGF ) induced increased proliferation in these cells whereas FGF 2 ( bFGF ), TGF-beta 1, and TGF-beta 2 inhibited proliferative activity
Hernández et al., Endocrinology 1995 : Vasopressin amplified basic fibroblast growth factor and epidermal growth factor inductions when used at submaximal doses
Mothe et al., Mol Biol Cell 1993 : We have studied the effects of nerve growth factor (NGF) and basic fibroblast growth factor (bFGF) on epidermal growth factor (EGF) binding to PC12 cells ... We show that NGF and bFGF rapidly induce a reduction in 125I-EGF binding to PC12 cells in a dose dependent manner
Yamagata et al., Clin Exp Pharmacol Physiol 1995 (Cerebrovascular Disorders...) : EGF induced greater DNA synthesis in SHRSP and SHRSR astrocytes compared with WKY astrocytes, although FGF had little or no effect