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SOS1 — ST8SIA2

Text-mined interactions from Literome

Aertsen et al., Appl Environ Microbiol 2005 : In addition, we found that the intrinsic pressure resistance of two types of Stx phages was very different, with one type surviving relatively well treatments of up to 400 MPa for 15 min at 20 degrees C. Interestingly, and in contrast to UV irradiation or mitomycin C treatment, pressure was not able to induce Stx prophage or an SOS response in several natural Stx producing STEC isolates
Toshima et al., Appl Environ Microbiol 2007 : We believe that Stx prophages are induced by the SOS response to DNA damage caused by DNase colicins, thus leading to higher Stx production
Nassar et al., Foodborne Pathog Dis 2013 : Recommended subinhibitory concentrations ( sub-MICs ) of azithromycin (AZI), gentamicin ( GEN ), imipenem ( IMI ), and rifampicin ( RIF ) were evaluated in comparison to norfloxacin ( NOR ), an SOS-inducer, to assess the role of the SOS response in Stx release ... For both strains at both sub-MICs, NOR and AZI caused SOS induced Stx production ( high recA, Q, and stx2 gene expression and high Stx2 production ), so they should be avoided in E. coli O157 : H7 treatment ; however, sub-MICs of RIF and IMI induced Stx2 production in an SOS independent manner except for one strain at the first twofold dilution below MIC of RIF where Stx2 production decreased