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IL8 — MMP3
Text-mined interactions from Literome
Meisser et al., Mol Hum Reprod 1999
:
The aim of this study was to determine the
effects of tumour necrosis factor alpha (TNF),
interleukin-1 alpha (IL-1alpha), macrophage colony stimulating factor ( MCSF ) and transforming growth factor beta ( TGFbeta ) on the secretion of
matrix metalloproteinases ( MMP ) , human chorionic gonadotrophin ( HCG ) and fetal fibronectin ( fFN ) by purified first trimester cytotrophoblastic cells (CTB) in vitro
Watanabe et al., Oral Oncol 2002
(Carcinoma, Squamous Cell...) :
Second, we thus also investigated the
effect of
IL-8 on
MMP release in OSCC cells
Bamba et al., J Gastroenterol 2003
:
Matrix metalloproteinase-3 secretion from human colonic subepithelial myofibroblasts :
role of
interleukin-17
Fleenor et al., Invest Ophthalmol Vis Sci 2003
:
In the trabecular meshwork ( TM ),
interleukin (IL)-1alpha is a potent
inducer of
MMP-3 expression
Sun et al., Connect Tissue Res 2003
(Arthritis, Rheumatoid) :
Furthermore, the
induction of the
MMP expression and activities by
interleukin-1 was suppressed by the oscillatory shear
Borghaei et al., Biochem Biophys Res Commun 2004
:
A 5T/6T polymorphic site in the matrix metalloproteinase-3 (MMP-3) promoter has been identified as a repressor element involved in inhibiting
induction of
MMP-3 transcription by
interleukin 1 ; and the 6T allele has been associated with decreased expression of MMP-3 as compared to the 5T allele
Mulayim et al., Fertil Steril 2004
:
To evaluate the
effect of
interleukin-8 (IL-8) on endometrial stromal cell metalloproteinase (
MMP ) activity and invasiveness ...
IL-8 increases
MMP activity and invasive capability of endometrial stromal cells in culture ; IL-8 may be involved in the pathogenesis of endometriosis and menstrual physiology
Inoue et al., Rheumatol Int 2005
(Osteoarthritis, Hip...) :
Induction of vascular endothelial growth factor and
matrix metalloproteinase-3 ( stromelysin ) by
interleukin-1 in human articular chondrocytes and synoviocytes
So et al., Gynecol Oncol 2004
(Neoplasm Invasiveness...) :
IL-8 did not
affect cellular migration,
MMP2 activation, or uPA expression
Stewart et al., BMC molecular biology 2007
:
Interleukin-4 inhibition of
interleukin-1 induced expression of
matrix metalloproteinase-3 (MMP-3) is independent of lipoxygenase and PPARgamma activation in human gingival fibroblasts
Chang et al., Rheumatol Int 2007
(Chondrosarcoma) :
Activin A suppresses
interleukin-1 induced
matrix metalloproteinase 3 secretion in human chondrosarcoma cells
Hanrahan et al., J Clin Oncol 2010
(Lung Neoplasms) :
For example,
increases in
IL-8 with VCP,
MMP-9 with CP, and VEGF with vandetanib monotherapy were associated with increased progression risk, and increase in intercellular adhesion molecule 1 with vandetanib was associated with decreased risk
Wang et al., J Biol Chem 2010
:
Importance of protein-tyrosine phosphatase-alpha catalytic domains for interactions with SHP-2 and
interleukin-1 induced
matrix metalloproteinase-3 expression
de Launay et al., Am J Pathol 2010
(Arthritis, Experimental...) :
Constitutively active mutants of each Ras protein
enhanced IL-1ß induced FLS
matrix metalloproteinase-3 production, while only active H-Ras enhanced
IL-8 production
Gotoh et al., Connect Tissue Res 2013
:
Extracellular ß-NAD ( + ) inhibits
interleukin-1 induced
matrix metalloproteinase-1 and -3 expression on human gingival fibroblasts
Thirkettle et al., J Biol Chem 2013
(Breast Neoplasms) :
In particular,
MMP-8 proteolytically
activates IL-8 and, thereby, regulates neutrophil chemotaxis in vivo