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FLT1 — SETD2

Text-mined interactions from Literome

Tipoe et al., Respir Physiol Neurobiol 2003 (Anoxia) : Hence, HIF-1 may activate the expression of VEGF and VEGFR-1 in the CB and the expression of VEGF in the chemoreceptors may play a paracrine role in the vascular remodeling during chronic hypoxia
Okuyama et al., J Biol Chem 2006 : Thus, loss-of-function and gain-of-function approaches demonstrated that HIF-1 activity is necessary and sufficient for basal and hypoxia induced VEGFR1 expression in bone marrow derived MSC
Hwang et al., Yonsei Med J 2013 : Induction of HIF-1a and TUBB3 in AGS is VEGFR-1 mediated and PlGF dependent
Hinkel et al., Curr Vasc Pharmacol 2013 (Constriction, Pathologic...) : Hif-1a , a master regulator of ischemia-responsive gene induction, controls pro-angiogenic gene expression of VEGF-A, flt-1 , IGF-1 and erythropoietin, rendering its overexpression an attractive tool for therapeutic neovascularization