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FOS — IL13
Text-mined interactions from Literome
Seppänen et al., Oncol Res 1998
(Adenocarcinoma...) :
In the present study, we have investigated the
effects of interferons-alpha (IFN-alpha) and -gamma ( IFN-gamma ),
interleukin-10 (IL-10) and -13 ( IL-13 ), transforming growth factor-beta1 ( TGF-beta1 ), granulocyte-macrophage colony stimulating factor ( GM-CSF ), and tumor necrosis factor-alpha (TNF-alpha) on cell proliferation and induction of transcription factors
AP-1 and NF-kappaB in UM-EC-3 human endometrial adenocarcinoma cells and UT-OC-5 ovarian carcinoma cells in vitro
Liacini et al., Matrix Biol 2002
(Osteoarthritis, Hip) :
Inhibition of
interleukin-1 stimulated MAP kinases,
activating protein-1 (AP-1) and nuclear factor kappa B (NF-kappa B) transcription factors down-regulates matrix metalloproteinase gene expression in articular chondrocytes
Li et al., Zhonghua Shao Shang Za Zhi 2004
(Acute Lung Injury) :
The pulmonary
AP-1 activity
increased with the enhanced expression of
IL-13 , which was related to the development of SIRS-ALI
Masuda et al., J Immunol 2004
:
The results of the present study have shown that direct interaction between
AP-1 and GATA proteins
plays an important role in
IL-13 transcription in mast cells
Wang et al., Nat Immunol 2006
:
Tumor necrosis factor receptor associated factor 6 ( TRAF6 ) is critical for mediating Toll-like receptor ( TLR )
-interleukin 1 receptor (IL-1R) signaling and subsequent
activation of NF-kappaB and
AP-1 , transcriptional activators of innate immunity
Shimamura et al., Clin Cancer Res 2010
(Adenocarcinoma...) :
c-Jun and
c-Fos of the AP-1 family of nuclear factors were
activated by
IL-13 only in IL-13Ralpha2 positive cells
Fujisawa et al., Int J Cancer 2012
(Neoplasm Invasiveness...) :
Mechanistically,
IL-13 enhanced ERK1/2,
AP-1 and MMP activities only in IL-13Ra2 positive cells but not in IL-13Ra2 negative cells
Sung et al., J Biol Chem 1993
:
Stimulation of
interleukin-1 gene transcription may be
caused by the stimulation of transcription factor activities, including those of
AP-1 , by these protein phosphatase inhibitors
Swiergiel et al., Brain Res Bull 1996
:
The role of cerebral noradrenergic systems in the
Fos response to
interleukin-1
Chang et al., Brain Res 1996
:
FOS expression
induced by
interleukin-1 or acute morphine treatment in the rat hypothalamus is attenuated by chronic exposure to morphine
Manna et al., J Immunol 1998
:
TNF induced activation of another nuclear transcription factor,
AP-1 , was
suppressed by
IL-13