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EGF — POLDIP2

Text-mined interactions from Literome

Douziech et al., Cell Signal 1999 (Pancreatic Neoplasms) : In MIA PaCa-2 cells, EGF and bombesin stimulated the in gel activation of p38 while p44/p42 kinases exhibited high basal activity and no response to stimuli
Wu et al., DNA Cell Biol 1999 (Brain Neoplasms...) : Basal and EGF induced JNK and p38 MAPK kinase activities were equivalent in parental cancer cells and EGFR inhibited subclones
Thoresen et al., Cell Physiol Biochem 2003 : EGF receptor stimulation also transiently activated p38 , and inhibition of p38 by SB203580 markedly decreased the DNA synthesis
Guo et al., World J Gastroenterol 2005 : Pro-apoptotic TGF-beta1, anti-apoptotic TPA and EGF induce the phosphorylation of p38 through different mechanisms that can be distinguished by SB202190
Pham et al., J Cell Physiol 2008 : Ang II and EGF induced transient phosphorylation of ERK, p38 ( MAPK ) and CREB
Shaharabany et al., FEBS J 2008 : WNK4 augmented the phosphorylation of ERK1/2 and p38 in response to both hypertonicity and EGF
LaMarca et al., Hum Reprod 2008 : EGF induced the phosphorylation of the phosphatidylinositol 3-kinase (PI3-K) dependent proteins, Akt and GSK-3beta as well as both p42/44 MAPK and p38 mitogen activated protein kinases ( MAPK )
Iwayama et al., Nephron extra 2011 : Epidermal growth factor (EGF) activated ERK and p38MAPK but not JNK