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CCND1 — HDAC3

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: HDAC3 — CCND1 (physical association, affinity chromatography technology) Petre-Draviam et al., Oncogene 2005*
• IRef Biogrid Interaction: HDAC3 — CCND1 (physical association, affinity chromatography technology) Lin et al., J Biol Chem 2002*
• IRef Biogrid Interaction: HDAC3 — CCND1 (physical association, affinity chromatography technology) Fu et al., J Biol Chem 2005*
• IRef Biogrid Interaction: HDAC3 — CCND1 (colocalization, imaging technique) Fu et al., J Biol Chem 2005*
• IRef Biogrid Interaction: HDAC3 — CCND1 (physical association, affinity chromatography technology) Batra et al., Mol Cancer Ther 2010*
• IRef Hprd Interaction: Complex of 17 proteins (in vivo) Liu et al., Biochem J 2006
• IRef Hprd Interaction: HDAC3 — CCND1 (in vivo) Lin et al., J Biol Chem 2002*

Text-mined interactions from Literome

Peterson et al., Mol Cell Biol 2007 (Breast Neoplasms) : SMRT is required for full expression of the ERalpha target genes cyclin D1 , BCL-2, and progesterone receptor but not pS2, and its depletion significantly attenuated estrogen dependent proliferation of MCF-7 cells
Song et al., J Biol Chem 2008 : Overexpression of SMRT and NCoR attenuated the transcription of beta-catenin-TCF4-specific reporter gene and of CCND1 , an endogenous beta-catenin target gene