◀ Back to IL8

IL13 — IL8

Text-mined interactions from Literome

Kolios et al., Eur J Immunol 1999 : We have previously demonstrated that IL-13 enhances IL-1alpha induced IL-8 secretion by the colonic epithelial cell line HT-29
Stríz et al., Inflammation 1999 : The present study was designed to explore the role of the Th2 cytokine IL-4 and the functionally related interleukins IL-10, and IL-13 on the regulation of IL-8 release by HBECs ... IL-13 did not affect the release of IL-8 induced by TNF ( 111 +/- 9 pM )
Bonecchi et al., J Immunol 2000 (Dermatitis, Atopic...) : Induction of functional IL-8 receptors by IL-4 and IL-13 in human monocytes
Shim et al., Am J Physiol Lung Cell Mol Physiol 2001 (Metaplasia) : Control airway epithelium contained few leukocytes, but intratracheal instillation of IL-13 resulted in time dependent leukocyte recruitment by IL-13 induced IL-8-like chemoattractant expression in airway epithelium
Yoshida et al., Invest Ophthalmol Vis Sci 2001 : IL-4 and -13 , but not IL-10, enhanced constitutive and TNF-alpha induced HRPE IL-8 and MCP-1 secretion ... IL-13 potentiated TNF-alpha induced MCP-1, but not IL-8 secretion by cocultures
Lottaz et al., J Invest Dermatol 2001 : We hypothesize that inhibition of this pathway prevents secretion of interleukin-1, thereby downregulating interleukin-1 dependent autocrine induction of interleukin-8
Thomas et al., Pediatr Res 2002 : IL-4 caused modest inhibition and IL-10 and IL-13 caused no inhibition of IL-8 secretion in all groups
Prause et al., Eur J Pharmacol 2003 : Pharmacological modulation of interleukin-17 induced GCP-2-, GRO-alpha- and interleukin-8 release in human bronchial epithelial cells ... We found that interleukin-17 ( 1-1000 ng/ml ) increased the release of GCP-2, GRO-alpha and interleukin-8 in a concentration dependent manner
Nasu et al., Fertil Steril 2003 : The effects of IL-13 and TNF-beta, on the production of IL-6, IL-11, leukemia inhibitory factor (LIF), IL-8 , growth regulated oncogene alpha ( GROalpha ), monocyte chemoattractant protein-1 ( MCP-1 ), regulated on activation, T-cell expressed and secreted ( RANTES ), and eotaxin were investigated
Waehre et al., J Am Coll Cardiol 2003 (Coronary Artery Disease) : Our main findings were : 1 ) gene expression of several chemokines ( i.e., macrophage inflammatory protein [MIP ] -1alpha, MIP-1beta, and interleukin [ IL]-8 ) and chemokine receptors ( i.e., CC chemokine receptor [ CCR]1, CCR2, CCR4, and CCR5 ) was markedly increased among CAD patients compared with healthy control subjects ; 2 ) treatment with atorvastatin and simvastatin markedly reduced the mRNA levels of some of these chemokines ( i.e., MIP-1alpha, MIP-1beta, IL-8 ) and receptors ( i.e., CCR1 and CCR2 ), with the most pronounced effect in the atorvastatin group ; and 3 ) statin therapy reduced the spontaneous release of IL-8 and MIP-1alpha from PBMCs in CAD patients
Wright et al., J Clin Immunol 2003 (Osteomyelitis...) : We investigated the potential down-regulators of chemokine secretion IL-4, IL-10, IL-13 , dexamethasone, and PGE2, IL-10 and IL-13 down-regulate M. tuberculosis induced IL-8 , IP-10, RANTES, and MCP-1 secretion from MG-63 cells by between 48 and 94 % ( P < 0.05 ) but do not inhibit chemokine gene transcription
Vittori et al., J Allergy Clin Immunol 1992 : Protective effect of nedocromil sodium on the interleukin-1 induced production of interleukin-8 in human bronchial epithelial cells
Uchiyama et al., Am J Obstet Gynecol 1992 : These results indicate that interleukin-8-like chemotactic factor participates in the cervical ripening at term pregnancy and that the production of this factor is controlled effectively by interleukin-1
Soloff et al., Endocrinology 2004 : In situ analysis of interleukin-1 induced transcription of cox-2 and il-8 in cultured human myometrial cells
Wuyts et al., J Heart Lung Transplant 2004 : N-acetylcysteine inhibits interleukin-17 induced interleukin-8 production from human airway smooth muscle cells : a possible role for anti-oxidative treatment in chronic lung rejection ?
Melk et al., Transplantation 2004 (Growth Disorders...) : During rhGH therapy in children with transplants, interleukin (IL)-2 production increased threefold at 4 weeks, and IL-4 and IL-13 increased by 70 % at 12 weeks
Hirota et al., Rheumatol Int 1992 : Production of interleukin 8 by cultured synovial cells in response to interleukin 1 and tumor necrosis factor
Hashmi et al., Coron Artery Dis 2006 (Angina, Unstable) : Role of interleukin-17 and interleukin-17 induced cytokines interleukin-6 and interleukin-8 in unstable coronary artery disease
Haber et al., Exp Parasitol 2009 (Hyperalgesia...) : Leishmania major : interleukin-13 increases the infection induced hyperalgesia and the levels of interleukin-1beta and interleukin-12 in rats
Darbonne et al., J Clin Invest 1991 : Their binding of IL-8 is rapidly reversible and does not result in receptor internalization, although bound IL-8 is resistant to extraction by pH 3 buffer at 5 degrees C. 125I-IL-8 binding to red cells was not inhibited by epidermal growth factor or interleukin 1, but was inhibited by monocyte chemotactic peptide-1, which is not a neutrophil chemotaxin, but is a member of the same family of polypeptides as IL-8
Larsen et al., Biochem Biophys Res Commun 1991 : 1,25 ( OH ) 2-D3 is a potent regulator of interleukin-1 induced interleukin-8 expression and production
Hua et al., Molecular oral microbiology 2010 (Periodontal Diseases) : Furthermore, as little as 2 µg ml(-1) mPE was sufficient to inhibit interleukin-1ß induced secretion of interleukin-8 in both gingival epithelial cells and THP-1 cells
Vindeløv et al., Growth Horm IGF Res 2011 (Adenoma...) : Interleukin-8 production from human somatotroph adenoma cells is stimulated by interleukin-1ß and inhibited by growth hormone releasing hormone and somatostatin
Strieter et al., Am J Respir Cell Mol Biol 1990 : The induction of AMO derived IL-8 by tumor necrosis factor (TNF), lipopolysaccharide (LPS), and interleukin-1 ( IL-1 beta ) was shown to be both dose and time dependent
Zhu et al., Journal of biomedical research 2010 : Recombinant IL-13 inhibited tumor necrosis factor-a (TNF-a), IL-1a, IL-1ß, monocyte chemoattractant protein-1 ( MCP-1 ), IL-8 , and transforming growth factor-ß1 ( TGF-ß1 ) mRNA expressions in a dose dependent manner
de Waal Malefyt et al., J Immunol 1993 : Furthermore, IL-13 inhibited production of IL-1 alpha, IL-1 beta, IL-6, IL-8 , IL-10, IL-12 p35, IL-12 p40, macrophage inflammatory protein-1 alpha, granulocyte/macrophage-CSF, granulocyte-CSF, IFN-alpha, and TNF alpha by monocytes activated with LPS
Ng et al., J Biol Chem 1994 : Although tumor necrosis factor (TNF) and interleukin-1 ( IL ) induce the expression of the IL-8 , TNF stimulated gene 6, and plasminogen activation inhibitor-2 genes and the activation of NF-kappa B with nearly identical kinetics, the two cytokines differ significantly in the induction of the IL-6 gene in all primary fibroblasts tested
Seitz et al., Eur J Immunol 1996 : In resting and cytokine stimulated MNC, IL-13 markedly inhibited IL-1 beta, IL-8 and monocyte chemoattractant protein-1 ( MCP-1 ) release and potently stimulated interleukin-1 receptor antagonist ( IL-1ra ) synthesis
Kolios et al., Br J Pharmacol 1996 : 4. HT-29 cells produced IL-8 following stimulation with IL-1 alpha or TNF-alpha in a time- and a concentration dependent manner, while IFN-gamma, IL-10 and IL-13 did not induce IL-8 production by HT-29 cells ... 6. Experiments using cycloheximide demonstrated that this synergistic effect of IL-13 and IL-1 alpha on IL-8 secretion was not through de novo protein synthesis
Hedges et al., Cytokine 1996 : IL-5, IL-12, IL-13 and TGF-beta 1 additively enhanced the bacterially induced IL-6 secretion, but they did not affect IL-8 secretion
Jordan et al., Br J Pharmacol 1997 : IL-13 , but not IL-10, significantly enhanced IL-8 and MCP-1 release in response to IL-1alpha or TNFalpha
Gronert et al., J Exp Med 1998 : Identification of a human enterocyte lipoxin A4 receptor that is regulated by interleukin (IL)-13 and interferon gamma and inhibits tumor necrosis factor alpha induced IL-8 release
Krakauer et al., J Infect Dis 1998 : Interleukin-8 production by human monocytic cells in response to staphylococcal exotoxins is direct and independent of interleukin-1 and tumor necrosis factor-alpha