◀ Back to ABL1
ABL1 — IL6
Text-mined interactions from Literome
Park et al., Exp Hematol 2010
(Blast Crisis...) :
In addition,
interleukin-3 induced
c-ABL phosphorylation and constitutively enhanced extracellular signal regulated kinase phosphorylation was not inhibited by imatinib in SNUCML-02
Reynaud et al., Cancer Cell 2011
(Leukemia, Myelogenous, Chronic, BCR-ABL Positive) :
We show that
BCR/ABL activity
controls Il-6 expression thereby establishing a paracrine feedback loop that sustains CML development
Matsuguchi et al., J Biol Chem 1994
(Leukemia, Megakaryoblastic, Acute) :
Shc phosphorylation in myeloid cells is regulated by granulocyte macrophage colony stimulating factor,
interleukin-3 , and steel factor and is constitutively
increased by
p210BCR/ABL