◀ Back to NOS3

NOS3 — RNF19A

Text-mined interactions from Literome

Chen et al., Mol Pharmacol 1999 : p38 but not p44/42 mitogen activated protein kinase is required for nitric oxide synthase induction mediated by lipopolysaccharide in RAW 264.7 macrophages
Ajizian et al., J Infect Dis 1999 : Specific inhibitors of p38 and extracellular signal regulated kinase mitogen activated protein kinase pathways block inducible nitric oxide synthase and tumor necrosis factor accumulation in murine macrophages stimulated with lipopolysaccharide and interferon-gamma
Souza et al., Mol Med 2001 (MAP Kinase Signaling System) : Interestingly, however, SB203580, a known p38 inhibitor, completely inhibited arsenite induced phosphorylation of AKT and eNOS
Monier et al., J Infect Dis 2002 : Differential effects of p38- and extracellular signal regulated kinase mitogen activated protein kinase inhibitors on inducible nitric oxide synthase and tumor necrosis factor production in murine macrophages stimulated with Streptococcus pneumoniae
Guo et al., Di Yi Jun Yi Da Xue Xue Bao 2003 : Regulation of inducible nitricoxide synthase gene transcription by p38 mitogen activated protein kinase in human embryonic kidney 293 cells
Chen et al., Blood 2008 (MAP Kinase Signaling System) : Antioxidant seleno-l-methionine ; chemical inhibitors of p38 , ERK1/2, and mitochondrial complex II ; as well as dominant negative mutant forms of IkappaBalpha and NOX4 effectively blocked sCD40L induced eNOS down-regulation in HCAECs
Yamawaki et al., Biochem Biophys Res Commun 2012 (Inflammation) : The effect is mediated via inhibiting activation of NF-?B and p38 through stimulation of Akt/eNOS signaling and NO production
Ou et al., J Appl Physiol 2013 : In addition, exposure to oxLDL resulted in reduced AMPK mediated Akt/endothelial nitric oxide ( NO ) synthase signaling and the induction of phosphorylation of p38 mitogen activated protein kinase, which, in turn, activated NF-?B mediated inflammatory responses, such as the release of interleukin-8, the expression of the adhesion molecule, and the adherence of monocytic cells to human umbilical vein endothelial cells