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CTNNB1 — FBXW11
Pathways - manually collected, often from reviews:
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
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IRef Biogrid Interaction:
CTNNB1
—
FBXW11
(physical association, affinity chromatography technology)
Gao et al., Mol Cell 2011
-
IRef Biogrid Interaction:
CTNNB1
—
FBXW11
(physical association, affinity chromatography technology)
Watanabe et al., Proc Natl Acad Sci U S A 2005*
-
IRef Biogrid Interaction:
CTNNB1
—
FBXW11
(physical association, affinity chromatography technology)
Fuchs et al., Oncogene 1999
-
IRef Intact Interaction:
Complex of GSK3B-AXIN1-CTNNB1-FBXW11-GSK3B-AXIN1-CTNNB1-FBXW11
(association, anti bait coimmunoprecipitation)
Li et al., Cell 2012
-
IRef Intact Interaction:
FBXW11
—
CTNNB1
(physical association, anti bait coimmunoprecipitation)
Li et al., Cell 2012
-
IRef Intact Interaction:
FBXW11
—
CTNNB1
(physical association, anti tag coimmunoprecipitation)
Li et al., Cell 2012
Text-mined interactions from Literome
Spiegelman et al., Oncogene 2002
:
In the NIH3T3 cells, which express HOS, but not betaTrCP,
Wnt/beta-catenin signaling
leads to inhibition of
HOS promoter activity and NF-kappaB-driven transcription as well as to stabilization of beta-catenin
Katoh et al., Cancer Biol Ther 2006
:
Because GSK3beta dependent phosphorylation of
beta-catenin and SNAIL
leads to
FBXW1 ( betaTRCP ) -mediated ubiquitination and degradation, GSK3beta downregulation results in the stabilization and the nuclear accumulation of beta-catenin and SNAIL