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CA2 — CALR

Text-mined interactions from Literome

Li et al., Biol Pharm Bull 2003 : Over-expression of calreticulin enhanced the activities of 47 kDa Ca2+ dependent protein kinase (CDPK) that had been induced by cold treatment
Treves et al., Biochem J 1990 : Because of its properties and intraluminal location, calreticulin might play a critical role in Ca2+ storage and release in non-muscle cells, similar to that played by calsequestrin in the muscle sarcoplasmic reticulum
Camacho et al., Cell 1995 : We examined the role of the luminal Ca ( 2+ ) -binding protein calreticulin (CRT) in IP3 mediated Ca2+ signaling by using Ca2+ wave activity as a sensitive Ca2+ release assay
Nigam et al., J Biol Chem 1994 : The release of grp94, protein-disulfide isomerase, ERp72, calreticulin , and p50 was stimulated by Ca2+ in the presence of ATP
Liu et al., J Biol Chem 1997 : Overexpressing the ER Ca2+ binding protein calreticulin prevented IDAM induced cell death, the rise in cytosolic Ca2+ , and oxidative stress
Liu et al., J Biol Chem 1998 : Interestingly, neither prior ER stress nor calreticulin expression prevented lipid peroxidation, but both blocked the rise in intracellular free Ca2+ after TBHP treatment
Llewelyn Roderick et al., Cell Calcium 1998 (Calcium Signaling) : Using transiently expressed aequorin as a Ca2+ indicator in these cells, we investigated the role of calreticulin in intracellular Ca2+ storage, IP3 mediated Ca2+ release, and capacitative Ca2+ entry