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CA2 — PI3

Text-mined interactions from Literome

Li et al., Eur J Immunol 2000 (MAP Kinase Signaling System) : In analyses of signaling intermediates, the phosphatidylinositol 3-kinase (PI3K) inhibitor wortmannin partially suppressed the release of Ca2+ induced by coligation of CD19 and mIgM but the selective PKC inhibitor bisindolylmaleimide I ( BIM-I ) did not
Yang et al., Nat Neurosci 2001 (Synaptic Transmission) : However, neither stimulation of Ca2+ release from intracellular stores by photolysis of caged IP3, nor expression of a constitutively active phosphoinositide-3 kinase (PI3K*) in presynaptic motoneurons alone is sufficient to enhance transmission
Bierne et al., Cell Microbiol 2000 (Listeriosis) : Purified InlB stimulates association of PLC-gamma1 with one or more tyrosine phosphorylated proteins, followed by a transient increase in intracellular inositol 1,4,5-trisphosphate ( IP3 ) levels and a release of intracellular Ca2+ in a PI 3-kinase dependent manner
Rocic et al., Am J Physiol Cell Physiol 2003 (MAP Kinase Signaling System) : This effect was blocked by inhibitors of the ERK1/2 and p38 mitogen activated protein ( MAP ) kinase pathways, but not by PI3-kinase inhibition, and was dependent on PKC, intracellular Ca2+ , and tyrosine kinases
Thors et al., FEBS Lett 2004 : Inhibition of the Ca2+/calmodulin dependent protein kinase II or phosphoinositide 3-kinase (PI3K) had no effect
Garofalo et al., Proc Biol Sci 2009 : Using the same bioassay, we now demonstrate that this effect is confirmed in the presence of the exogenous NO donor S-nitroso-N-acetyl penicillamine, is independent from endocardial endothelium and guanylate cyclase/cGMP/protein kinase G and cAMP/protein kinase A pathways, involves a PI(3)kinase mediated activation of endothelial NO synthase and a modulation of the SR-CA(2+)ATPase ( SERCA2a ) pumps
Yamagata et al., Microvasc Res 2012 (Atherosclerosis) : IL-1ß decreased the expression of Ca2+/calmodulin dependent kinase II ( CaMKII ), eNOS, PON-1, phosphatidylinositol 3-kinase (PI3K) , PSD-95/Dlg/ZO-1 ( PZK1 ), and liver kinase B1 (LKB1)
Ahmed et al., J Biol Chem 1995 : Thus, stimulation of fMLP receptors gave rise to dual effects, activation of PI 3-kinase and intracellular Ca2+ mobilization ; both effects were necessary for the fMLP induced respiratory burst
Buhl et al., J Exp Med 1997 (Mast-Cell Sarcoma) : Antigen induced CD19 dependent PI3-kinase activation is required for normal phosphoinositide hydrolysis and Ca2+ mobilization responses