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IL13 — SOAT1

Text-mined interactions from Literome

Guiter et al., Blood 2004 (Lymphoma, B-Cell...) : IL-4 and IL-13 transcripts were absent in PMBL cell lines and expressed at low levels in tumors, indicating that, contrary to classical Hodgkin lymphoma ( cHL ), STAT6 activation is not due to an autocrine IL-4/IL-13 secretion
Bian et al., Proc Natl Acad Sci U S A 2005 : We now demonstrate that ( i ) an IL-4 receptor alpha-subunit ( IL-4Ralpha ) /Stat6 dependent but T cell independent pathway is the key for the nematode induced host NOS-2 inhibition ; ( ii ) endogenous IL-4 and IL-13 , the only known IL-4Ralpha ligands, are not required for activating the pathway ; and ( iii ) treatment of RAW264.7 cells with parasite cultured medium inhibits NOS-2 expression but not cyclooxygenase 2 expression
Chiba et al., J Smooth Muscle Res 2009 (Asthma...) : In the present study, the effects of interleukin-13 (IL-13) and IL-4 on activation of these STAT molecules were examined in cultured human bronchial smooth muscle cells ( hBSMCs )
Goenka et al., Immunol Res 2011 : Among the seven STAT proteins, STAT6 is activated by IL-4 and IL-13 and plays a predominant role in the immune system
Russell et al., Islets 2013 : Differential effects of interleukin-13 and interleukin-6 on Jak/STAT signaling and cell viability in pancreatic ß-cells
Seidel et al., Proc Natl Acad Sci U S A 1995 (Carcinoma, Hepatocellular...) : We have systematically examined the effects of the spacing between the TT and AA core half sites on the binding of the STAT complexes activated by IFN-gamma, interleukin (IL) 6, granulocyte-macrophage colony stimulating factor, and IL-4
Murata et al., J Immunol 1996 (Colonic Neoplasms) : Both IL-13 and IL-4 induced phosphorylation of IL-4 STAT ( STAT6 ) but not STAT1, STAT3, or STAT5