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ATP5O — NGF

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Inoue et al., J Neurochem 1988 (Adrenal Gland Neoplasms...) : As a consequence, during differentiation of PC12h cells to neuron-like cells, NGF increases the alpha form of Na+, K+-ATPase , but does not induce the alpha ( + ) form of the enzyme, which has a high sensitivity for cardiotonic steroid and is a characteristic form in neurons
Krakovskiĭ et al., Vestn Akad Med Nauk SSSR 1989 (Burns...) : Changes in the location of cAMP dependent protein kinase caused by these factors, the effect of the nerve growth factor on the activities of protein kinase and high-affinity ATPase , and the mechanism of antiproliferative action of staphylococcal enterotoxin A were specified
Morita et al., Cell Struct Funct 1988 (Pheochromocytoma) : Nerve growth factor (NGF) induced the activities of acetylcholinesterase (AChE) and Na+, K+-ATPase concomitant with neurite outgrowth in PC12h cells, while dibutyryl cyclic AMP ( DBcAMP ) caused the induction of AChE activity and neurite outgrowth but not Na+, K+-ATPase activity
Colangelo et al., Brain Res Mol Brain Res 1996 (Glioma) : Gel mobility shift assays using an oligonucleotide homologous to the AP-1 responsive element of the rat NGF gene ( AP-1NGF ) revealed that 12-O-tetradecanoyl phorbol-13-acetate ( TPA ) and, to a lesser extent, isoproterenol ( ISO ) and thapsigargin, a microsomal Ca ( 2+ ) -ATPase inhibitor, stimulated binding to AP-1NGF within 2 h