◀ Back to HNRNPH1

CCR2 — HNRNPH1

Text-mined interactions from Literome

Qu et al., J Exp Med 2004 : However, absence of CCR2 led to an increased labeling of the minor Gr-1int monocyte population, and the number of latex+ DCs that emigrated to LNs was correspondingly increased
Salazar-Gonzalez et al., Immunity 2006 (Salmonella Infections) : Thus, CCR6 dependent regulation of DCs is responsible for localized T cell dependent defense against entero-invasive pathogens
Robays et al., J Immunol 2007 (Asthma) : Using this approach, we show that CCR2 , but not CCR5 or CCR6, directly controls the accumulation of DCs into allergic lungs
Ibarra et al., Immunobiology 2011 (Arthritis, Experimental...) : To examine the mechanism by which DCs contribute to the increased susceptibility of arthritis in Ccr2?/? mice, we tested the hypothesis that CD8a? DCs are protective ( tolerogenic ) against autoimmune arthritis by examining the role of CD8a? DCs in Ccr2?/? and SKG mice
Qiu et al., J Immunol 2012 (Cryptococcosis...) : TLR9 deletion diminished 1 ) accumulation and activation of CD11b ( + ) dendritic cells (DCs) , 2 ) the induction of IFN-? and CCR2 chemokines CCL7, CCL12, but not CCL2, at week 1, and 3 ) pulmonary accumulation and activation of the major effector cells CD4 ( + ) and CD8 ( + ) T cells, CD11b ( + ) lung DCs, and exudate macrophages at week 3