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ACACA — CPT2

Text-mined interactions from Literome

Yamagishi et al., J Biol Chem 2001 : Leptin induced ROS generation, CPT-1 activation, ACC inhibition , and MCP-1 overproduction were found to be completely prevented by either genistein, a tyrosine kinase inhibitor, H-89, a protein kinase A (PKA) inhibitor, or tetradecylglycidate, a CPT-1 inhibitor
Guo et al., Lipids in health and disease 2012 (Calcium Signaling) : Furthermore, Cy-3-g substantially induced AMPK downstream target ACC phosphorylation and inactivation, and then decreased malonyl CoA contents, leading to stimulation of CPT-1 expression and significant increase of fatty acid oxidation in HepG2 cells
Lopaschuk et al., J Biol Chem 1994 : We therefore addressed the hypothesis that a decrease in acetyl-CoA carboxylase (ACC) activity and/or malonyl-CoA inhibition of CPT1 could account for the increase in the ability of the heart to oxidize fatty acids following birth