◀ Back to ANG

ANG — AVP

Text-mined interactions from Literome

Wu et al., Acta Pharmacol Sin 2000 (Anoxia) : Ang II enhanced AVP release, from ( 5.7 +/- 1.6 ) ng/ME in control decreasing to ( 2.6 +/- 1.2 ) ng/ME ( P < 0.05 ), meanwhile plasma corticosterone concentration was also increased markedly, from ( 356 +/- 58 ) in control to ( 536 +/- 134 ) micrograms/L plasma ( P < 0.05 ), which was partly abolished by administration of AVP antiserum
Gohlke et al., Naunyn Schmiedebergs Arch Pharmacol 2002 : In the brain, angiotensin II (Ang II) induces various effects such as blood pressure increase, the release of arginine vasopressin (AVP) and drinking behaviour
Shi et al., Am J Physiol Endocrinol Metab 2003 : The effect of intravenous angiotensin II (ANG II) on fetal brain c-fos expression and arginine vasopressin (AVP) release was studied in the near-term ovine fetus
Jain et al., Neuroendocrinology 2004 : As angiotensin II (Ang II) regulates CRH, AVP and sympathetic activity, we studied the expression of Ang II receptors in the hypothalamus and adrenal gland of GR transgenic and wild-type mice, adrenal catecholamines and mRNA for their rate limiting enzyme, tyrosine hydroxylase (TH)
Osanai et al., Hypertension 1992 (Hypertension) : Activation of phospholipase C by angiotensin II (Ang II) , thromboxane A2, arginine vasopressin , and endothelin-1 was compared in cultured glomerular mesangial cells and mesenteric vascular smooth muscle cells taken from 13- to 14-week-old hypertensive and normotensive Wistar-Kyoto rats ( blood pressure, 185 +/- 1 versus 135 +/- 2 mm Hg )
Shi et al., Peptides 2006 : However, few data exist regarding central ANG receptor mechanisms mediated AVP response during fetal life
Vallotton et al., J Cardiovasc Pharmacol 1990 : Ang II induced a 15-fold increase and AVP induced a fivefold increase of 6-keto-PGF1 alpha at 10 ( -6 ) M
Shi et al., Am J Physiol Endocrinol Metab 2010 : Intracerebroventricular Ang I significantly increased fetal blood pressure and c-fos expression in the supraoptic nuclei (SON) and the paraventricular nuclei ( PVN ) in the hypothalamus, accompanied by an increase of fetal plasma arginine vasopressin (AVP)
Wuthrich et al., Am J Physiol 1986 : AVP and BK were active on medullary cells, and ANG II was active mainly on cortical cells
Schiavone et al., Proc Natl Acad Sci U S A 1988 : At 0.04 microM, Ang II or Ang- ( 1-7 ) did not stimulate AVP release ... Neither Ang II nor Ang- ( 1-7 ) ( 4 microM ) caused a further enhancement of AVP release in the presence of [ Sar1,Thr8 ] Ang II
Moriguchi et al., Am J Physiol 1994 : To evaluate the hypothalamic vasopressin axis in the TG rat, we measured the central nervous system concentrations of AVP and determined the effect of angiotensin II (ANG II) and its NH2-terminal heptapeptide [ angiotensin- ( 1-7 ) ] on blood pressure, heart rate, and AVP release using brain microdialysis ... The plasma AVP responses to ANG II and ANG- ( 1-7 ) were comparable in the control and TG rats
Hogarty et al., Brain Res 1994 : We have previously shown that AT1 and AT2 angiotensin II (Ang II) receptors mediate the release of arginine vasopressin (AVP) to central injections of Ang II ... In this study we have tested the hypothesis that Ang II, acting at AT1 and AT2 receptors in the brain, is involved in mediating osmotically stimulated AVP release ... We conclude that brain Ang II receptors are involved in osmotically stimulated AVP release
Qadri et al., J Pharmacol Exp Ther 1993 : In addition, ANG II AT1 receptors in the SON can contribute to AVP release after periventricular ANG II receptor stimulation
Walker et al., J Appl Physiol 1995 (Acute Disease...) : We conclude that during acute hypercapnia of sufficient magnitude, and perhaps duration, AVP inhibits an ANG II-mediated stimulation of metabolism and respiration
Saydoff et al., Am J Physiol 1996 : Central serotonin ( 5-HT ) and angiotensin (ANG II) stimulate arginine vasopressin (AVP) , oxytocin ( OT ), and adrenocorticotropin ( ACTH ) secretion and increase blood pressure
Chiodera et al., Neuropeptides 1998 : The present study was undertaken in order to establish the possible involvement of melatonin in the mechanisms underlying the arginine-vasopressin (AVP) responses to physical exercise and angiotensin II (ANG II) ... These data indicate an involvement of melatonin in the mechanism underlying the AVP response to physical exercise, but not ANG II, in normal men
Chiodera et al., Metabolism 1998 : This study was performed to determine whether the stimulatory effect of plasma angiotensin II (ANG II) on arginine vasopressin (AVP) and oxytocin ( OT ) secretion in humans is mediated by AT1 subtype receptors