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NFKBIA — SMAD3

Text-mined interactions from Literome

Yamazaki et al., J Biol Chem 2009 : Furthermore, this p65 mediated inhibition of BMP2- and Smad-responsive promoter activity was restored after inhibition of NF-kappaB by the overexpression of the dominant negative IkappaBalpha
Smythies et al., J Biol Chem 2010 (Inflammation) : Inflammation anergy in human intestinal macrophages is due to Smad induced IkappaBalpha expression and NF-kappaB inactivation ... Likely contributing to this unique functional profile, intestinal macrophages expressed markedly down-regulated adapter proteins MyD88 and Toll interleukin receptor 1 domain containing adapter inducing interferon beta, which together mediate all TLR MyD88 dependent and -independent NF-kappaB signaling, did not phosphorylate NF-kappaB p65 or Smad induced IkappaBalpha , and did not translocate NF-kappaB into the nucleus