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CRK — E2F1

Text-mined interactions from Literome

Hershko et al., J Biol Chem 2006 : Both ASK1 and Wip1 are essential mediators of the E2F-p38 connection : knock down of ASK1 inhibits E2F1 induced phosphorylation of p38, whereas knock down of Wip1 prolongs E2F1 induced p38 phosphorylation
Hassan et al., Hepatology 2009 (Hepatitis C, Chronic...) : In addition, regulated expression of core protein in HepG2 or Huh7 cells was found to induce expression and activation of the transcription factor E2F1 and apoptosis signal regulating kinase 1 ( ASK1 ), activation of c-jun-N-terminal kinase ( JNK ) and p38 , and extracellular regulated kinase (ERK), and transcription factors activator protein 1 (AP-1), activating transcription factor 2 ( ATF-2 ), cyclic adenosine monophosphate response element binding ( CREB ), E2F1, hypoxia inducing factor 1 alpha ( HIF-1alpha ), and specificity protein 1
Lee et al., Mol Cells 2011 (Breast Neoplasms) : These results suggest that Pin1 overexpression and subsequent VEGF production in TAMR-MCF-7 cells are mediated through PI3-kinase or p38 kinase dependent E2F1 activation