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CCL2 — TGFB2

Text-mined interactions from Literome

Lee et al., American journal of physiology. Renal physiology 2009 (Albuminuria...) : Only transforming growth factor-beta ( TGF-beta ) significantly increased MCP-1 production, which was prevented by SB431542 and LY294002, indicating that signaling proceeded through the TGF-beta type I receptor kinase and the phosphatidylinositol 3-kinase pathway
Hembruff et al., Neoplasia (New York, N.Y.) 2010 (Disease Progression...) : Loss of transforming growth factor-beta signaling in mammary fibroblasts enhances CCL2 secretion to promote mammary tumor progression through macrophage dependent and -independent mechanisms
Zhu et al., J Bone Miner Res 1994 : The effects of interleukin-1 beta (IL-1 beta), tumor necrosis factor alpha (TNF-alpha), interleukin-6 (IL-6), transforming growth factor beta ( TGF-beta ), platelet derived growth factor ( PDGF-BB ), parathyroid hormone (PTH), and 1,25 ( OH ) 2D3 on MCP-1 expression in human osteoblastic cells were compared
Tesch et al., J Am Soc Nephrol 1998 (Glomerulonephritis) : However, the addition of recombinant MIF to mesangial cells did not affect mesangial cell proliferation or constitutive transforming growth factor-beta mRNA expression, nor did MIF induce monocyte chemoattractant protein-1 mRNA expression
Weiss et al., J Neuroimmunol 1998 : Astrocyte expression of monocyte chemoattractant protein-1 is differentially regulated by transforming growth factor beta