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CSF1 — IRF6

Text-mined interactions from Literome

Kamthong et al., Biochem J 2000 : Expression of M-CSF was inducible by interleukin-1alpha (IL-1alpha), lipopolysaccharide (LPS) and PMA as demonstrated by a biological activity assay, Northern-blot analysis and reverse transcriptase ( RT ) PCR ... Treatment of the cells with forskolin or dibutyryl-cAMP attenuated the expression of M-CSF induced by IL-1alpha or LPS , but not by PMA
Gruber et al., J Immunol 1992 : LPS and IL-1 beta, but not IL-6 or TNF-alpha, dramatically augmented the ability of anti-CD45, anti-CD44, and anti-LFA-3 antibodies to induce M-CSF secretion but failed to stimulate M-CSF secretion in the absence of antibody
Sester et al., Immunobiology 2005 : Using toll-like receptor (tlr)9 as a model CSF-1 repressed gene, we show that LPS induced tlr9 expression in BMM only when CSF-1 was present, suggesting that LPS relieves CSF-1 mediated inhibition to induce gene expression ... By real time PCR we confirmed that the expression of a selection of these genes, including integral membrane protein 2B (itm2b), receptor activity modifying protein 2 ( ramp2 ) and macrophage-specific gene 1 (mpg-1), were repressed by CSF-1 and were induced by LPS only in the presence of CSF-1
Tanaka et al., Life Sci 2006 : The present study was undertaken to determine the effect of nicotine and LPS on the expression of macrophage colony stimulating factor ( M-CSF ), osteoprotegerin (OPG), and prostaglandin E2 ( PGE2 ) in osteoblasts, and the indirect effect of nicotine and LPS on the formation of osteoclast-like cells
Shoji et al., Acta Biochim Biophys Sin (Shanghai) 2007 : We determined the effect of nicotine and LPS on alkaline phosphatase ( ALPase ) activity, PGE ( 2 ) production, and the expression of cyclooxygenase ( COX-1, COX-2 ), PGE ( 2 ) receptors Ep1 > 4, and macrophage colony stimulating factor ( M-CSF ) in human osteoblastic Saos-2 cells
Bonfield et al., J Immunol 2008 : Overexpression of PPARgamma prevented LPS stimulated M-CSF production in RAW 264.7 cells, an effect that was abrogated by a specific PPARgamma antagonist, GW9662 ... Use of proteasome inhibitor, MG-132 or a PPARgamma agonist, pioglitazone, prevented LPS mediated M-CSF induction
Seo et al., Bone 2010 (MAP Kinase Signaling System...) : Osteoblasts exhibit innate immune response by expressing M-CSF in response to lipopolysaccharide (LPS) ... LPS induced M-CSF expression is mediated by ERK
Li et al., Oncogene 2011 (Neoplasms) : Here, we showed that serum amyloid-A (SAA) and cathelicidin ( LL-37 ) stimulated M-CSF and MCP-1 expression with or without lipopolysaccharide (LPS) administration ; conversely, lipoxin-A ( 4 ) ( LXA ( 4 ) ) and annexin-A1 (ANXA1) inhibited LPS induced M-CSF and MCP-1 production by human ( HepG2 ) and mouse ( H22 ) hepatocellular carcinoma cells (HCCs)
Chodakewitz et al., J Immunol 1990 : Furthermore, production of keratinocyte derived M-CSF is increased by bacterial LPS
Qin et al., J Immunol 2012 (Inflammation) : The M1 proinflammatory phenotype is induced by LPS , IFN-?, and GM-CSF, and IL-4, IL-13, and M-CSF induce anti-inflammatory M2 macrophages
Becker et al., J Leukoc Biol 1989 : In contrast to the AMO, monocyte production of CSF-1 was enhanced by LPS
Chen et al., J Immunol 1983 : LPS , however, causes minimal or no inhibition of 125I-CSF-1 binding by PEM from LPS-resistant C3H/HeJ mice
Hamilton et al., J Leukoc Biol 1994 : In the presence of LPS , both M-CSF and GM-CSF were up-regulated by cyclooxygenase inhibition ( indomethacin, 10 ( -5 ) M ), while G-CSF was down-regulated
Xie et al., Br J Haematol 1993 (Neoplasms, Experimental) : In PU5/1.8 cells, CSF-1 binding was already very low and was not further down-regulated, but c-fos and TNF mRNA was inducible by LPS
Chen et al., Exp Hematol 1993 : The loss of M-CSF receptors induced by LPS can be inhibited by neomycin and compound 48/80, two potent phospholipase C (PLC) inhibitors, but not by phospholipase A2, calpain, protein kinase C ( PKC ) or protease inhibitors
Furman et al., J Neurosci Res 1996 (Glioma) : However, when 1,25- ( OH ) 2D3 is used in combination with LPS, a partial reduction in LPS induced levels of M-CSF and TNF-alpha mRNA is observed
Steube et al., Leuk Res 1997 : Vice versa, secretion of macrophage CSF (M-CSF) could be induced by TPA, but not by LPS ... However, LPS enhanced the TPA induced M-CSF production