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ICAM1 — SOD2

Text-mined interactions from Literome

Morandini et al., Cell Mol Biol Incl Cyto Enzymol 1999 (Melanoma...) : Specific inhibition of Cu/Zn-SOD activity by DTIC ( diethyldithiocarbamic acid ), in presence of Paraquat or TNF, was followed by an upregulation of ICAM-1 expression ( 60 and 20 %, respectively )
Konturek et al., Scand J Gastroenterol 2000 (Reperfusion Injury...) : 1 ) exposure to I/R produces gastric lesions mediated by the excessive formation of free radicals, resulting in suppression of both gastric microcirculation and secretory activity of the stomach ; 2 ) SOD and allopurinol accelerate the healing of I/R lesions, probably due to suppression of oxygen free radicals and improvement of gastric microcirculation ; and 3 ) the upregulation of SOD mRNA, with subsequent increase in the SOD production and local release of IL-1beta and TNF-alpha, may activate ICAM-1 expression and neutrophil infiltration, which appear to play an important role in the progression of I/R induced acute gastric erosions into chronic ulcers
Chen et al., J Pharmacol Exp Ther 2003 : In addition, expression of superoxide dismutase by adenovirus suppressed TNF-alpha induced VCAM-1, E-selectin, and ICAM-1 mRNA accumulation
Rajapakse et al., Am J Physiol Heart Circ Physiol 2009 (Hyperglycemia...) : In the cells cultured under a low-glucose condition when no increased HBP flux occurred, azaserine enhanced the manganese-superoxide dismutase ( MnSOD ) protein level and also inhibited the oxidative stress and the expression of VCAM-1 and ICAM-1 in response to TNF-alpha
Kong et al., Lipids in health and disease 2013 : However, comparing to the model rabbits, levels of TNF-a, IL-1ß and MDA decreased significantly and serum SOD activity increased , gene and protein expressions of E-sel, ICAM-1 , VCAM-1 and MCP-1 in aortas decreased significantly with the treatment of kaempferol ... However, comparing to the model rabbits, levels of TNF-a, IL-1ß and MDA decreased significantly and serum SOD activity increased , gene and protein expressions of E-sel, ICAM-1 , VCAM-1 and MCP-1 in aortas decreased significantly with the treatment of kaempferol
Hiromatsu et al., Immunology 1993 (Graves Disease) : Neither catalase nor superoxide dismutase , which are free-radical scavengers, inhibited the expression of ICAM-1 on thyroid cells
Arai et al., Biochem J 1998 (Second Messenger Systems) : TNF-alpha upregulation of endothelial-cell ICAM-1 expression was inhibited by the cell-permeable antioxidants, or by the adenovirus mediated intracellular overexpression of Cu, Zn-superoxide dismutase , but not by the exogenous ( extracellular ) administration of the cell-impermeable antioxidants, superoxide dismutase and/or catalase
Bhunia et al., J Biol Chem 1998 : Overexpression of endogeneous CuZn-superoxide dismutase via an adenoviral vector carrying cDNA for CuZn-superoxide dismutase, also inhibited LacCer induced ICAM-1 expression in endothelial cells