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CNTF — STAT1

Text-mined interactions from Literome

Zhang et al., Exp Eye Res 2005 : Retinas from embryonic and neonatal stages showed that STAT3 but not STAT1 was activated in response to ciliary neurotrophic factor (CNTF) , leukemia inhibitory factor (LIF), fibroblast growth factor-1 (FGF1), fibroblast growth factor-2 (FGF2), epidermal growth factor (EGF), interferon-alpha ( IFN-alpha ) and interferon-gamma (IFN-gamma) in distinct patterns
Monroe et al., Free Radic Biol Med 2006 : CdCl2 concentrations as low as 0.1 microM and for times as brief as 2 h significantly reduced CNTF induced tyrosine phosphorylation of both STAT1 and STAT3, the principle substrates of Jak kinases in neurons
Rezende et al., J Biol Chem 2012 (Diabetes Mellitus, Experimental) : CNTF increased STAT3 phosphorylation in WT and SOCS3kd mice and MIN6 cells but reduced STAT1 phosphorylation only in WT mice, in contrast to streptozotocin and IL1ß
Rajan et al., J Neurosci Res 1996 : In cultures of embryonic rat septum and other brain areas, in addition to Stat3, CNTF also activates Stat1