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CCL20 — JUN

Text-mined interactions from Literome

Hosokawa et al., Clin Exp Immunol 2005 : On the other hand, we found that not only NF-kappaB, p38 MAPK and ERK but also c-Jun NH2-terminal kinase (JNK) are involved in CCL20 production induced by E. coli LPS
Kanda et al., Eur J Immunol 2009 (Psoriasis) : CCL20 production induced by PRL plus IL-17 was suppressed by antisense c-Fos, c-Jun , p50, and p65
Hosokawa et al., Cell Immunol 2013 (Periodontitis) : Polyinosinic-polycytidylic acid ( Poly I:C ), which is a TLR3 agonist, stimulation could moderately induce CCL20 production in HGFs. Poly I:C synergistically enhanced CCL20 expression from IL-1ß stimulated HGFs. Inhibitors of p38 MAPK, extracellular signal regulated kinase ( ERK ), c-Jun N terminal kinase (JNK) , and NF-?B significantly inhibited CCL20 production in Poly I:C/IL-1ß stimulated HGFs. Western blot analysis disclosed phosphorylation of p38 MAPK, JNK, and I?B-a were enhanced in Poly I:C/IL-1ß treated HGFs