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GAL — IKBKG

Text-mined interactions from Literome

Garbati et al., Gene Expr 2008 : Ser484 and Ser494 in REL are the major sites of IKK phosphorylation in vitro : evidence that IKK does not directly enhance GAL4-REL transactivation ... We also show that the previously reported effects of overexpressed IKK and tumor necrosis factor treatment on GAL4-REL transactivation are due to IKK mediated activation of the endogenous NF-kappa B pathway, which increases transcription from kappa B sites in the promoter of a commonly used GAL4 expression vector
Lu et al., Proc Natl Acad Sci U S A 2010 (Disease Models, Animal...) : CYM2503 potentiated the galanin stimulated IP1 accumulation in HEK293 cells stably expressing GalR2 receptor, whereas it exhibited no detectable affinity for the ( 125 ) I galanin binding site of GalR2 receptor, an effect consistent with that of a positive allosteric modulator